1,802 research outputs found

    A Morphological Associative Memory Employing A Stored Pattern Independent Kernel Image and Its Hardware Model

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    An associative memory provides a convenient way for pattern retrieval and restoration, which has an important role for handling data distorted with noise. As an effective associative memory, we paid attention to a morphological associative memory (MAM) proposed by Ritter. The model is superior to ordinary associative memory models in terms of calculation amount, memory capacity, and perfect recall rate. However, in general, the kernel design becomes difficult as the stored pattern increases because the kernel uses a part of each stored pattern. In this paper, we propose a stored pattern independent kernel design method for the MAM and design the MAM employing the proposed kernel design with a standard digital manner in parallel architecture for acceleration. We confirm the validity of the proposed kernel design method by auto- and hetero-association experiments and investigate the efficiency of the hardware acceleration. A high-speed operation (more than 150 times in comparison with software execution) is achieved in the custom hardware. The proposed model works as an intelligent pre-processor for the Brain-Inspired Systems (Brain-IS) working in real world

    MATLAB PROGRAM CODES FOR BIDIRECTIONAL ASSOCIATIVE MEMORY NETWORKS

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    Objective Neural networks are being used for solving problems in various diverse areas including education, research, business, management, and many more. In this article, models describing the dynamics of bidirectional associative memory (BAM) neural networks are considered. Methods: MATLAB, the numerical computing environment and programming language is used for solving certain problems associated with BAM. Results: The concept of BAM networks is improved so that it can be applied to a wider class of networks. Algorithm for solving BAM problems is studied. And also the MATLAB program codes to find the weight matrix, to test the net with input, and to generate activation functions are accompanied. Conclusion: MATLAB programming can be effectively used to solve the problems associated with BAM

    The malleable brain: plasticity of neural circuits and behavior: A review from students to students

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    One of the most intriguing features of the brain is its ability to be malleable, allowing it to adapt continually to changes in the environment. Specific neuronal activity patterns drive long-lasting increases or decreases in the strength of synaptic connections, referred to as long-term potentiation (LTP) and long-term depression (LTD) respectively. Such phenomena have been described in a variety of model organisms, which are used to study molecular, structural, and functional aspects of synaptic plasticity. This review originated from the first International Society for Neurochemistry (ISN) and Journal of Neurochemistry (JNC) Flagship School held in Alpbach, Austria (Sep 2016), and will use its curriculum and discussions as a framework to review some of the current knowledge in the field of synaptic plasticity. First, we describe the role of plasticity during development and the persistent changes of neural circuitry occurring when sensory input is altered during critical developmental stages. We then outline the signaling cascades resulting in the synthesis of new plasticity-related proteins, which ultimately enable sustained changes in synaptic strength. Going beyond the traditional understanding of synaptic plasticity conceptualized by LTP and LTD, we discuss system-wide modifications and recently unveiled homeostatic mechanisms, such as synaptic scaling. Finally, we describe the neural circuits and synaptic plasticity mechanisms driving associative memory and motor learning. Evidence summarized in this review provides a current view of synaptic plasticity in its various forms, offers new insights into the underlying mechanisms and behavioral relevance, and provides directions for future research in the field of synaptic plasticity.Fil: Schaefer, Natascha. University of Wuerzburg; AlemaniaFil: Rotermund, Carola. University of Tuebingen; AlemaniaFil: Blumrich, Eva Maria. Universitat Bremen; AlemaniaFil: Lourenco, Mychael V.. Universidade Federal do Rio de Janeiro; BrasilFil: Joshi, Pooja. Robert Debre Hospital; FranciaFil: Hegemann, Regina U.. University of Otago; Nueva ZelandaFil: Jamwal, Sumit. ISF College of Pharmacy; IndiaFil: Ali, Nilufar. Augusta University; Estados UnidosFil: García Romero, Ezra Michelet. Universidad Veracruzana; MéxicoFil: Sharma, Sorabh. Birla Institute of Technology and Science; IndiaFil: Ghosh, Shampa. Indian Council of Medical Research; IndiaFil: Sinha, Jitendra K.. Indian Council of Medical Research; IndiaFil: Loke, Hannah. Hudson Institute of Medical Research; AustraliaFil: Jain, Vishal. Defence Institute of Physiology and Allied Sciences; IndiaFil: Lepeta, Katarzyna. Polish Academy of Sciences; ArgentinaFil: Salamian, Ahmad. Polish Academy of Sciences; ArgentinaFil: Sharma, Mahima. Polish Academy of Sciences; ArgentinaFil: Golpich, Mojtaba. University Kebangsaan Malaysia Medical Centre; MalasiaFil: Nawrotek, Katarzyna. University Of Lodz; ArgentinaFil: Paid, Ramesh K.. Indian Institute of Chemical Biology; IndiaFil: Shahidzadeh, Sheila M.. Syracuse University; Estados UnidosFil: Piermartiri, Tetsade. Universidade Federal de Santa Catarina; BrasilFil: Amini, Elham. University Kebangsaan Malaysia Medical Centre; MalasiaFil: Pastor, Verónica. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Biología Celular y Neurociencia ; ArgentinaFil: Wilson, Yvette. University of Melbourne; AustraliaFil: Adeniyi, Philip A.. Afe Babalola University; NigeriaFil: Datusalia, Ashok K.. National Brain Research Centre; IndiaFil: Vafadari, Benham. Polish Academy of Sciences; ArgentinaFil: Saini, Vedangana. University of Nebraska; Estados UnidosFil: Suárez Pozos, Edna. Instituto Politécnico Nacional; MéxicoFil: Kushwah, Neetu. Defence Institute of Physiology and Allied Sciences; IndiaFil: Fontanet, Paula. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Biología Celular y Neurociencia ; ArgentinaFil: Turner, Anthony J.. University of Leeds; Reino Unid

    From Bidirectional Associative Memory to a noise-tolerant, robust Protein Processor Associative Memory

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    AbstractProtein Processor Associative Memory (PPAM) is a novel architecture for learning associations incrementally and online and performing fast, reliable, scalable hetero-associative recall. This paper presents a comparison of the PPAM with the Bidirectional Associative Memory (BAM), both with Kosko's original training algorithm and also with the more popular Pseudo-Relaxation Learning Algorithm for BAM (PRLAB). It also compares the PPAM with a more recent associative memory architecture called SOIAM. Results of training for object-avoidance are presented from simulations using player/stage and are verified by actual implementations on the E-Puck mobile robot. Finally, we show how the PPAM is capable of achieving an increase in performance without using the typical weighted-sum arithmetic operations or indeed any arithmetic operations

    Financial distress prediction using the hybrid associative memory with translation

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    This paper presents an alternative technique for financial distress prediction systems. The method is based on a type of neural network, which is called hybrid associative memory with translation. While many different neural network architectures have successfully been used to predict credit risk and corporate failure, the power of associative memories for financial decision-making has not been explored in any depth as yet. The performance of the hybrid associative memory with translation is compared to four traditional neural networks, a support vector machine and a logistic regression model in terms of their prediction capabilities. The experimental results over nine real-life data sets show that the associative memory here proposed constitutes an appropriate solution for bankruptcy and credit risk prediction, performing significantly better than the rest of models under class imbalance and data overlapping conditions in terms of the true positive rate and the geometric mean of true positive and true negative rates.This work has partially been supported by the Mexican CONACYT through the Postdoctoral Fellowship Program [232167], the Spanish Ministry of Economy [TIN2013-46522-P], the Generalitat Valenciana [PROMETEOII/2014/062] and the Mexican PRODEP [DSA/103.5/15/7004]. We would like to thank the Reviewers for their valuable comments and suggestions, which have helped to improve the quality of this paper substantially

    Maintenance of aversive memories shown by fear extinction-impaired phenotypes is associated with increased activity in the amygdaloid-prefrontal circuit

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    Although aversive memory has been mainly addressed by analysing the changes occurring in average populations, the study of neuronal mechanisms of outliers allows understanding the involvement of individual differences in fear conditioning and extinction. We recently developed an innovative experimental model of individual differences in approach and avoidance behaviors, classifying the mice as Approaching, Balancing or Avoiding animals according to their responses to conflicting stimuli. The approach and avoidance behaviors appear to be the primary reactions to rewarding and threatening stimuli and may represent predictors of vulnerability (or resilience) to fear. We submitted the three mice phenotypes to Contextual Fear Conditioning. In comparison to Balancing animals, Approaching and Avoiding mice exhibited no middle- or long-term fear extinction. The two non-extinguishing phenotypes exhibited potentiated glutamatergic neurotransmission (spontaneous excitatory postsynaptic currents/spinogenesis) of pyramidal neurons of medial prefrontal cortex and basolateral amygdala. Basing on the a priori individuation of outliers, we demonstrated that the maintenance of aversive memories is linked to increased spinogenesis and excitatory signaling in the amygdala-prefrontal cortex fear matri

    Fuzzy ART: Fast Stable Learning and Categorization of Analog Patterns by an Adaptive Resonance System

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    A Fuzzy ART model capable of rapid stable learning of recognition categories in response to arbitrary sequences of analog or binary input patterns is described. Fuzzy ART incorporates computations from fuzzy set theory into the ART 1 neural network, which learns to categorize only binary input patterns. The generalization to learning both analog and binary input patterns is achieved by replacing appearances of the intersection operator (n) in AHT 1 by the MIN operator (Λ) of fuzzy set theory. The MIN operator reduces to the intersection operator in the binary case. Category proliferation is prevented by normalizing input vectors at a preprocessing stage. A normalization procedure called complement coding leads to a symmetric theory in which the MIN operator (Λ) and the MAX operator (v) of fuzzy set theory play complementary roles. Complement coding uses on-cells and off-cells to represent the input pattern, and preserves individual feature amplitudes while normalizing the total on-cell/off-cell vector. Learning is stable because all adaptive weights can only decrease in time. Decreasing weights correspond to increasing sizes of category "boxes". Smaller vigilance values lead to larger category boxes. Learning stops when the input space is covered by boxes. With fast learning and a finite input set of arbitrary size and composition, learning stabilizes after just one presentation of each input pattern. A fast-commit slow-recode option combines fast learning with a forgetting rule that buffers system memory against noise. Using this option, rare events can be rapidly learned, yet previously learned memories are not rapidly erased in response to statistically unreliable input fluctuations.British Petroleum (89-A-1204); Defense Advanced Research Projects Agency (90-0083); National Science Foundation (IRI-90-00530); Air Force Office of Scientific Research (90-0175

    Associative learning on imbalanced environments: An empirical study

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    Associative memories have emerged as a powerful computational neural network model for several pattern classification problems. Like most traditional classifiers, these models assume that the classes share similar prior probabilities. However, in many real-life applications the ratios of prior probabilities between classes are extremely skewed. Although the literature has provided numerous studies that examine the performance degradation of renowned classifiers on different imbalanced scenarios, so far this effect has not been supported by a thorough empirical study in the context of associative memories. In this paper, we fix our attention on the applicability of the associative neural networks to the classification of imbalanced data. The key questions here addressed are whether these models perform better, the same or worse than other popular classifiers, how the level of imbalance affects their performance, and whether distinct resampling strategies produce a different impact on the associative memories. In order to answer these questions and gain further insight into the feasibility and efficiency of the associative memories, a large-scale experimental evaluation with 31 databases, seven classification models and four resampling algorithms is carried out here, along with a non-parametric statistical test to discover any significant differences between each pair of classifiers.This work has partially been supported by the Mexican Science and Technology Council (CONACYT-Mexico) through the Postdoctoral Fellowship Program (232167), the Mexican PRODEP(DSA/103.5/15/7004), the Spanish Ministry of Economy(TIN2013-46522-P) and the Generalitat Valenciana (PROMETEOII/2014/062)

    Reinstatement of long-term memory following erasure of its behavioral and synaptic expression in Aplysia.

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    Long-term memory (LTM) is believed to be stored in the brain as changes in synaptic connections. Here, we show that LTM storage and synaptic change can be dissociated. Cocultures of Aplysia sensory and motor neurons were trained with spaced pulses of serotonin, which induces long-term facilitation. Serotonin (5HT) triggered growth of new presynaptic varicosities, a synaptic mechanism of long-term sensitization. Following 5HT training, two antimnemonic treatments-reconsolidation blockade and inhibition of PKM--caused the number of presynaptic varicosities to revert to the original, pretraining value. Surprisingly, the final synaptic structure was not achieved by targeted retraction of the 5HT-induced varicosities but, rather, by an apparently arbitrary retraction of both 5HT-induced and original synapses. In addition, we find evidence that the LTM for sensitization persists covertly after its apparent elimination by the same antimnemonic treatments that erase learning-related synaptic growth. These results challenge the idea that stable synapses store long-term memories
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