836 research outputs found

    Immunization for complex network based on the effective degree of vertex

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    The basic idea of many effective immunization strategies is first to rank the importance of vertices according to the degrees of vertices and then remove the vertices from highest importance to lowest until the network becomes disconnected. Here we define the effective degrees of vertex, i.e., the number of its connections linking to un-immunized nodes in current network during the immunization procedure, to rank the importance of vertex, and modify these strategies by using the effective degrees of vertices. Simulations on both the scale-free network models with various degree correlations and two real networks have revealed that the immunization strategies based on the effective degrees are often more effective than those based on the degrees in the initial network.Comment: 16 pages, 5 figure

    Recoverable prevalence in growing scale-free networks and the effective immunization

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    We study the persistent recoverable prevalence and the extinction of computer viruses via e-mails on a growing scale-free network with new users, which structure is estimated form real data. The typical phenomenon is simulated in a realistic model with the probabilistic execution and detection of viruses. Moreover, the conditions of extinction by random and targeted immunizations for hubs are derived through bifurcation analysis for simpler models by using a mean-field approximation without the connectivity correlations. We can qualitatively understand the mechanisms of the spread in linearly growing scale-free networks.Comment: 9 pages, 9 figures, 1 table. Update version after helpful referee comment

    Epidemic processes in complex networks

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    In recent years the research community has accumulated overwhelming evidence for the emergence of complex and heterogeneous connectivity patterns in a wide range of biological and sociotechnical systems. The complex properties of real-world networks have a profound impact on the behavior of equilibrium and nonequilibrium phenomena occurring in various systems, and the study of epidemic spreading is central to our understanding of the unfolding of dynamical processes in complex networks. The theoretical analysis of epidemic spreading in heterogeneous networks requires the development of novel analytical frameworks, and it has produced results of conceptual and practical relevance. A coherent and comprehensive review of the vast research activity concerning epidemic processes is presented, detailing the successful theoretical approaches as well as making their limits and assumptions clear. Physicists, mathematicians, epidemiologists, computer, and social scientists share a common interest in studying epidemic spreading and rely on similar models for the description of the diffusion of pathogens, knowledge, and innovation. For this reason, while focusing on the main results and the paradigmatic models in infectious disease modeling, the major results concerning generalized social contagion processes are also presented. Finally, the research activity at the forefront in the study of epidemic spreading in coevolving, coupled, and time-varying networks is reported.Comment: 62 pages, 15 figures, final versio

    Immunization of Real Complex Communication Networks

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    Most communication networks are complex. In this paper, we address one of the fundamental problems we are facing nowadays, namely, how we can efficiently protect these networks. To this end, we study an immunization strategy and found that it works as good as targeted immunization, but using only local information about the network topology. Our findings are supported with numerical simulations of the Susceptible-Infected-Removed (SIR) model on top of real communication networks, where immune nodes are previously identified by a covering algorithm. The results provide useful hints in the way to design and deploying a digital immune system.Comment: 6 pages. To appear in the European Physical Journal B (2006

    Reactive immunization on complex networks

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    Epidemic spreading on complex networks depends on the topological structure as well as on the dynamical properties of the infection itself. Generally speaking, highly connected individuals play the role of hubs and are crucial to channel information across the network. On the other hand, static topological quantities measuring the connectivity structure are independent on the dynamical mechanisms of the infection. A natural question is therefore how to improve the topological analysis by some kind of dynamical information that may be extracted from the ongoing infection itself. In this spirit, we propose a novel vaccination scheme that exploits information from the details of the infection pattern at the moment when the vaccination strategy is applied. Numerical simulations of the infection process show that the proposed immunization strategy is effective and robust on a wide class of complex networks

    Dynamical patterns of epidemic outbreaks in complex heterogeneous networks

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    We present a thorough inspection of the dynamical behavior of epidemic phenomena in populations with complex and heterogeneous connectivity patterns. We show that the growth of the epidemic prevalence is virtually instantaneous in all networks characterized by diverging degree fluctuations, independently of the structure of the connectivity correlation functions characterizing the population network. By means of analytical and numerical results, we show that the outbreak time evolution follows a precise hierarchical dynamics. Once reached the most highly connected hubs, the infection pervades the network in a progressive cascade across smaller degree classes. Finally, we show the influence of the initial conditions and the relevance of statistical results in single case studies concerning heterogeneous networks. The emerging theoretical framework appears of general interest in view of the recently observed abundance of natural networks with complex topological features and might provide useful insights for the development of adaptive strategies aimed at epidemic containment.Comment: 13 pages, 11 figure

    Generalized Network Dismantling

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    Finding the set of nodes, which removed or (de)activated can stop the spread of (dis)information, contain an epidemic or disrupt the functioning of a corrupt/criminal organization is still one of the key challenges in network science. In this paper, we introduce the generalized network dismantling problem, which aims to find the set of nodes that, when removed from a network, results in a network fragmentation into subcritical network components at minimum cost. For unit costs, our formulation becomes equivalent to the standard network dismantling problem. Our non-unit cost generalization allows for the inclusion of topological cost functions related to node centrality and non-topological features such as the price, protection level or even social value of a node. In order to solve this optimization problem, we propose a method, which is based on the spectral properties of a novel node-weighted Laplacian operator. The proposed method is applicable to large-scale networks with millions of nodes. It outperforms current state-of-the-art methods and opens new directions in understanding the vulnerability and robustness of complex systems.Comment: 6 pages, 5 figure

    Velocity and hierarchical spread of epidemic outbreaks in scale-free networks

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    We study the effect of the connectivity pattern of complex networks on the propagation dynamics of epidemics. The growth time scale of outbreaks is inversely proportional to the network degree fluctuations, signaling that epidemics spread almost instantaneously in networks with scale-free degree distributions. This feature is associated with an epidemic propagation that follows a precise hierarchical dynamics. Once the highly connected hubs are reached, the infection pervades the network in a progressive cascade across smaller degree classes. The present results are relevant for the development of adaptive containment strategies.Comment: 4 pages, 4 figures, final versio
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