Dynamical Systems on Networks: A Tutorial

We give a tutorial for the study of dynamical systems on networks. We focus especially on "simple" situations that are tractable analytically, because they can be very insightful and provide useful springboards for the study of more complicated scenarios. We briefly motivate why examining dynamical systems on networks is interesting and important, and we then give several fascinating examples and discuss some theoretical results. We also briefly discuss dynamical systems on dynamical (i.e., time-dependent) networks, overview software implementations, and give an outlook on the field.Comment: 39 pages, 1 figure, submitted, more examples and discussion than original version, some reorganization and also more pointers to interesting direction

Long ties accelerate noisy threshold-based contagions

Network structure can affect when and how widely new ideas, products, and behaviors are adopted. In widely-used models of biological contagion, interventions that randomly rewire edges (generally making them "longer") accelerate spread. However, there are other models relevant to social contagion, such as those motivated by myopic best-response in games with strategic complements, in which an individual's behavior is described by a threshold number of adopting neighbors above which adoption occurs (i.e., complex contagions). Recent work has argued that highly clustered, rather than random, networks facilitate spread of these complex contagions. Here we show that minor modifications to this model, which make it more realistic, reverse this result: we allow very rare below-threshold adoption, i.e., rarely adoption occurs when there is only one adopting neighbor. To model the trade-off between long and short edges we consider networks that are the union of cycle-power-$k$ graphs and random graphs on $n$ nodes. Allowing adoptions below threshold to occur with order $1/\sqrt{n}$ probability along some "short" cycle edges is enough to ensure that random rewiring accelerates spread. Simulations illustrate the robustness of these results to other commonly-posited models for noisy best-response behavior. Hypothetical interventions that randomly rewire existing edges or add random edges (versus adding "short", triad-closing edges) in hundreds of empirical social networks reduce time to spread. This revised conclusion suggests that those wanting to increase spread should induce formation of long ties, rather than triad-closing ties. More generally, this highlights the importance of noise in game-theoretic analyses of behavior

The Majority Illusion in Social Networks

Social behaviors are often contagious, spreading through a population as individuals imitate the decisions and choices of others. A variety of global phenomena, from innovation adoption to the emergence of social norms and political movements, arise as a result of people following a simple local rule, such as copy what others are doing. However, individuals often lack global knowledge of the behaviors of others and must estimate them from the observations of their friends' behaviors. In some cases, the structure of the underlying social network can dramatically skew an individual's local observations, making a behavior appear far more common locally than it is globally. We trace the origins of this phenomenon, which we call "the majority illusion," to the friendship paradox in social networks. As a result of this paradox, a behavior that is globally rare may be systematically overrepresented in the local neighborhoods of many people, i.e., among their friends. Thus, the "majority illusion" may facilitate the spread of social contagions in networks and also explain why systematic biases in social perceptions, for example, of risky behavior, arise. Using synthetic and real-world networks, we explore how the "majority illusion" depends on network structure and develop a statistical model to calculate its magnitude in a network

Contagions in Random Networks with Overlapping Communities

We consider a threshold epidemic model on a clustered random graph with overlapping communities. In other words, our epidemic model is such that an individual becomes infected as soon as the proportion of her infected neighbors exceeds the threshold q of the epidemic. In our random graph model, each individual can belong to several communities. The distributions for the community sizes and the number of communities an individual belongs to are arbitrary. We consider the case where the epidemic starts from a single individual, and we prove a phase transition (when the parameter q of the model varies) for the appearance of a cascade, i.e. when the epidemic can be propagated to an infinite part of the population. More precisely, we show that our epidemic is entirely described by a multi-type (and alternating) branching process, and then we apply Sevastyanov's theorem about the phase transition of multi-type Galton-Watson branching processes. In addition, we compute the entries of the matrix whose largest eigenvalue gives the phase transition.Comment: Minor modifications for the second version: added comments (end of Section 3.2, beginning of Section 5.3); moved remark (end of Section 3.1, beginning of Section 4.1); corrected typos; changed titl