26,613 research outputs found

    Worker Retention, Response Quality, and Diversity in Microtask Crowdsourcing: An Experimental Investigation of the Potential for Priming Effects to Promote Project Goals

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    Online microtask crowdsourcing platforms act as efficient resources for delegating small units of work, gathering data, generating ideas, and more. Members of research and business communities have incorporated crowdsourcing into problem-solving processes. When human workers contribute to a crowdsourcing task, they are subject to various stimuli as a result of task design. Inter-task priming effects - through which work is nonconsciously, yet significantly, influenced by exposure to certain stimuli - have been shown to affect microtask crowdsourcing responses in a variety of ways. Instead of simply being wary of the potential for priming effects to skew results, task administrators can utilize proven priming procedures in order to promote project goals. In a series of three experiments conducted on Amazon’s Mechanical Turk, we investigated the effects of proposed priming treatments on worker retention, response quality, and response diversity. In our first two experiments, we studied the effect of initial response freedom on sustained worker participation and response quality. We expected that workers who were granted greater levels of freedom in an initial response would be stimulated to complete more work and deliver higher quality work than workers originally constrained in their initial response possibilities. We found no significant relationship between the initial response freedom granted to workers and the amount of optional work they completed. The degree of initial response freedom also did not have a significant impact on subsequent response quality. However, the influence of inter-task effects were evident based on response tendencies for different question types. We found evidence that consistency in task structure may play a stronger role in promoting response quality than proposed priming procedures. In our final experiment, we studied the influence of a group-level priming treatment on response diversity. Instead of varying task structure for different workers, we varied the degree of overlap in question content distributed to different workers in a group. We expected groups of workers that were exposed to more diverse preliminary question sets to offer greater diversity in response to a subsequent question. Although differences in response diversity were revealed, no consistent trend between question content overlap and response diversity prevailed. Nevertheless, combining consistent task structure with crowd-level priming procedures - to encourage diversity in inter-task effects across the crowd - offers an exciting path for future study

    User-Behavior Based Detection of Infection Onset

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    A major vector of computer infection is through exploiting software or design flaws in networked applications such as the browser. Malicious code can be fetched and executed on a victim’s machine without the user’s permission, as in drive-by download (DBD) attacks. In this paper, we describe a new tool called DeWare for detecting the onset of infection delivered through vulnerable applications. DeWare explores and enforces causal relationships between computer-related human behaviors and system properties, such as file-system access and process execution. Our tool can be used to provide real time protection of a personal computer, as well as for diagnosing and evaluating untrusted websites for forensic purposes. Besides the concrete DBD detection solution, we also formally define causal relationships between user actions and system events on a host. Identifying and enforcing correct causal relationships have important applications in realizing advanced and secure operating systems. We perform extensive experimental evaluation, including a user study with 21 participants, thousands of legitimate websites (for testing false alarms), as well as 84 malicious websites in the wild. Our results show that DeWare is able to correctly distinguish legitimate download events from unauthorized system events with a low false positive rate (< 1%)

    Neural connectivity in syntactic movement processing

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    Linguistic theory suggests non-canonical sentences subvert the dominant agent-verb-theme order in English via displacement of sentence constituents to argument (NP-movement) or non-argument positions (wh-movement). Both processes have been associated with the left inferior frontal gyrus and posterior superior temporal gyrus, but differences in neural activity and connectivity between movement types have not been investigated. In the current study, functional magnetic resonance imaging data were acquired from 21 adult participants during an auditory sentence-picture verification task using passive and active sentences contrasted to isolate NP-movement, and object- and subject-cleft sentences contrasted to isolate wh-movement. Then, functional magnetic resonance imaging data from regions common to both movement types were entered into a dynamic causal modeling analysis to examine effective connectivity for wh-movement and NP-movement. Results showed greater left inferior frontal gyrus activation for Wh > NP-movement, but no activation for NP > Wh-movement. Both types of movement elicited activity in the opercular part of the left inferior frontal gyrus, left posterior superior temporal gyrus, and left medial superior frontal gyrus. The dynamic causal modeling analyses indicated that neither movement type significantly modulated the connection from the left inferior frontal gyrus to the left posterior superior temporal gyrus, nor vice-versa, suggesting no connectivity differences between wh- and NP-movement. These findings support the idea that increased complexity of wh-structures, compared to sentences with NP-movement, requires greater engagement of cognitive resources via increased neural activity in the left inferior frontal gyrus, but both movement types engage similar neural networks.This work was supported by the NIH-NIDCD, Clinical Research Center Grant, P50DC012283 (PI: CT), and the Graduate Research Grant and School of Communication Graduate Ignition Grant from Northwestern University (awarded to EE). (P50DC012283 - NIH-NIDCD, Clinical Research Center Grant; Graduate Research Grant and School of Communication Graduate Ignition Grant from Northwestern University)Published versio

    Using the Literature to Identify Confounders

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    Prior work in causal modeling has focused primarily on learning graph structures and parameters to model data generating processes from observational or experimental data, while the focus of the literature-based discovery paradigm was to identify novel therapeutic hypotheses in publicly available knowledge. The critical contribution of this dissertation is to refashion the literature-based discovery paradigm as a means to populate causal models with relevant covariates to abet causal inference. In particular, this dissertation describes a generalizable framework for mapping from causal propositions in the literature to subgraphs populated by instantiated variables that reflect observational data. The observational data are those derived from electronic health records. The purpose of causal inference is to detect adverse drug event signals. The Principle of the Common Cause is exploited as a heuristic for a defeasible practical logic. The fundamental intuition is that improbable co-occurrences can be “explained away” with reference to a common cause, or confounder. Semantic constraints in literature-based discovery can be leveraged to identify such covariates. Further, the asymmetric semantic constraints of causal propositions map directly to the topology of causal graphs as directed edges. The hypothesis is that causal models conditioned on sets of such covariates will improve upon the performance of purely statistical techniques for detecting adverse drug event signals. By improving upon previous work in purely EHR-based pharmacovigilance, these results establish the utility of this scalable approach to automated causal inference
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