11,716 research outputs found

    SKOPE: A connectionist/symbolic architecture of spoken Korean processing

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    Spoken language processing requires speech and natural language integration. Moreover, spoken Korean calls for unique processing methodology due to its linguistic characteristics. This paper presents SKOPE, a connectionist/symbolic spoken Korean processing engine, which emphasizes that: 1) connectionist and symbolic techniques must be selectively applied according to their relative strength and weakness, and 2) the linguistic characteristics of Korean must be fully considered for phoneme recognition, speech and language integration, and morphological/syntactic processing. The design and implementation of SKOPE demonstrates how connectionist/symbolic hybrid architectures can be constructed for spoken agglutinative language processing. Also SKOPE presents many novel ideas for speech and language processing. The phoneme recognition, morphological analysis, and syntactic analysis experiments show that SKOPE is a viable approach for the spoken Korean processing.Comment: 8 pages, latex, use aaai.sty & aaai.bst, bibfile: nlpsp.bib, to be presented at IJCAI95 workshops on new approaches to learning for natural language processin

    Intake factors and intake processes in adult language learning

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    Deficits of knowledge versus executive control in semantic cognition: Insights from cued naming

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    Deficits of semantic cognition in semantic dementia and in aphasia consequent on CVA (stroke) are qualitatively different. Patients with semantic dementia are characterised by progressive degradation of central semantic representations, whereas multimodal semantic deficits in stroke aphasia reflect impairment of executive processes that help to direct and control semantic activation in a task-appropriate fashion [Jefferies, E., & Lambon Ralph, M. A. (2006). Semantic impairment in stroke aphasia vs. semantic dementia: A case-series comparison. Brain 129, 2132-2147]. We explored interactions between these two aspects of semantic cognition by examining the effects of cumulative phonemic cueing on picture naming in case series of these two types of patient. The stroke aphasic patients with multimodal semantic deficits cued very readily and demonstrated near-perfect name retrieval when cumulative phonemic cues reached or exceeded the target name's uniqueness point. Therefore, knowledge of the picture names was largely intact for the aphasic patients, but they were unable to retrieve this information without cues that helped to direct activation towards the target response. Equivalent phonemic cues engendered significant but much more limited benefit to the semantic dementia patients: their naming was still severely impaired even when most of the word had been provided. In contrast to the pattern in the stroke aphasia group, successful cueing was mainly confined to the more familiar un-named pictures. We propose that this limited cueing effect in semantic dementia follows from the fact that concepts deteriorate in a graded fashion [Rogers, T. T., Lambon Ralph, M. A., Garrard, P., Bozeat, S., McClelland, J. L., & Hodges, J. R., et al. (2004). The structure and deterioration of semantic memory: A neuropsychological and computational investigation. Psychological Review 111, 205-235]. For partially degraded items, the residual conceptual knowledge may be insufficient to drive speech production to completion but these items might reach threshold when they are bolstered by cues. (C) 2007 Elsevier Ltd. All rights reserved

    Methods of testing and diagnosing model error : dual and single route cascaded models of reading aloud

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    Models of visual word recognition have been assessed by both factorial and regression approaches. Factorial approaches tend to provide a relatively weak test of models, and regression approaches give little indication of the sources of models’ mispredictions, especially when parameters are not optimal. A new alternative method, involving regression on model error, combines these two approaches with parameter optimization. The method is illustrated with respect to the dual route cascaded model of reading aloud. In contrast to previous investigations, this method provides clear evidence that there are parameter-independent problems with the model, and identifies two specific sources of misprediction made by model

    Models of atypical development must also be models of normal development

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    Functional magnetic resonance imaging studies of developmental disorders and normal cognition that include children are becoming increasingly common and represent part of a newly expanding field of developmental cognitive neuroscience. These studies have illustrated the importance of the process of development in understanding brain mechanisms underlying cognition and including children ill the study of the etiology of developmental disorders

    Are developmental disorders like cases of adult brain damage? Implications from connectionist modelling

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    It is often assumed that similar domain-specific behavioural impairments found in cases of adult brain damage and developmental disorders correspond to similar underlying causes, and can serve as convergent evidence for the modular structure of the normal adult cognitive system. We argue that this correspondence is contingent on an unsupported assumption that atypical development can produce selective deficits while the rest of the system develops normally (Residual Normality), and that this assumption tends to bias data collection in the field. Based on a review of connectionist models of acquired and developmental disorders in the domains of reading and past tense, as well as on new simulations, we explore the computational viability of Residual Normality and the potential role of development in producing behavioural deficits. Simulations demonstrate that damage to a developmental model can produce very different effects depending on whether it occurs prior to or following the training process. Because developmental disorders typically involve damage prior to learning, we conclude that the developmental process is a key component of the explanation of endstate impairments in such disorders. Further simulations demonstrate that in simple connectionist learning systems, the assumption of Residual Normality is undermined by processes of compensation or alteration elsewhere in the system. We outline the precise computational conditions required for Residual Normality to hold in development, and suggest that in many cases it is an unlikely hypothesis. We conclude that in developmental disorders, inferences from behavioural deficits to underlying structure crucially depend on developmental conditions, and that the process of ontogenetic development cannot be ignored in constructing models of developmental disorders
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