MOESM7 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 7: Table S6. The distribution of AA usage in CDR3

MOESM4 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 4: Table S3. The distribution of V-J combinations in each sample

MOESM6 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 6: Table S5. The distribution of CDR3 AA clonotypes in each sample

MOESM5 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 5: Table S4. The distribution of V-D-J combinations in each sample

MOESM3 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 3: Table S2. The distribution of V and J gene segments in each sample

MOESM2 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 2: Table S1. The analysis of TCRβ immune repertoire sequencing data

MOESM1 of Peripheral T cell receptor beta immune repertoire is promptly reconstituted after acute myocardial infarction

Additional file 1: Fig. S1. Two-round nested amplicon arm-PCR for TCRβ immune repertoire. n = 5 per group. Fig. S2. The clonotypes analysis of TCRβ base sequence. a Quantification (plot grays) and frequencies (plot colors) of base sequence clonotypes. Light grays indicate the overlapping clonotypes, while dark grays indicate the total clonotypes per sample. b Comparison of total clonotypes of TCRβ base sequence. c The percentage of overlapping base sequence between healthy controls and AMI patients. Fig. S3. The distribution of CDR3 AA length between healthy controls and AMI patients. n = 5 per group