323 research outputs found

    deForm: An interactive malleable surface for capturing 2.5D arbitrary objects, tools and touch

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    We introduce a novel input device, deForm, that supports 2.5D touch gestures, tangible tools, and arbitrary objects through real-time structured light scanning of a malleable surface of interaction. DeForm captures high-resolution surface deformations and 2D grey-scale textures of a gel surface through a three-phase structured light 3D scanner. This technique can be combined with IR projection to allow for invisible capture, providing the opportunity for co-located visual feedback on the deformable surface. We describe methods for tracking fingers, whole hand gestures, and arbitrary tangible tools. We outline a method for physically encoding fiducial marker information in the height map of tangible tools. In addition, we describe a novel method for distinguishing between human touch and tangible tools, through capacitive sensing on top of the input surface. Finally we motivate our device through a number of sample applications

    Clark Family Letters - Letters from Edward Weston to his aunt Jane Clark 1865-1870

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    Letters written by Edward Weston to his aunt Jane Clark. Written from Maitland and Hythe by Edwards regarding visit to Mauriceton (1865), rent for land at the Lakes, parents' health, invitation for Jane to come and stay (May 1867); Kate - baby boy has cleft palate (June 1869). From the family papers of William & John Clark of Cluny, Bothwell 1812-1872-Royal Society Collection RS8/F9 Letters 1-

    Solstice: An Electronic Journal of Geography and Mathematics: Vol. 30, No. 2

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    https://deepblue.lib.umich.edu/bitstream/2027.42/152345/1/SolsticeVolumeXXXNumber2Final.pdfhttps://deepblue.lib.umich.edu/bitstream/2027.42/152345/4/SolsticeVolumeXXXNumber2Final.pdfDescription of SolsticeVolumeXXXNumber2.pdf : XXXDescription of SolsticeVolumeXXXNumber2Final.pdf : Solstice, Vol. XXIX, No. 2

    Financial Distress and Restructuring Models

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    In recent years, the literature of financial distress has been enriched by the development of formal models. This paper develops a synthesis of that formal analysis, linking it to related finance literature and corporate strategies for distressed financial restructuring. Several key assumptions generate different results which predict the effects of financial distress on investment efficiency and restructuring strategy. Central to these strategies are the recontracting arrangements proposed between owners, creditors and other relevant stakeholders. The critical factors in the alternative models are: (1) the term structure of the firm’s debt, (2) the role of the seniority of debts, (3) the effects of exchange offers, (4) the effects of an automatic stay on debt payments, and (5) the role of alternative voting rules

    Financial Distress and Restructuring Models

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    In recent years, the literature of financial distress has been enriched by the development of formal models. This paper develops a synthesis of that formal analysis, linking it to related finance literature and corporate strategies for distressed financial restructuring. Several key assumptions generate different results which predict the effects of financial distress on investment efficiency and restructuring strategy. Central to these strategies are the recontracting arrangements proposed between owners, creditors and other relevant stakeholders. The critical factors in the alternative models are: (1) the term structure of the firm’s debt, (2) the role of the seniority of debts, (3) the effects of exchange offers, (4) the effects of an automatic stay on debt payments, and (5) the role of alternative voting rules

    Lack of Rbl1/p107 Effects on Cell Proliferation and Maturation in the Inner Ear

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    Loss of postnatal mammalian auditory hair cells (HCs) is irreversible. Earlier studies have highlighted the importance of the Retinoblastoma family of proteins (pRBs) (i.e., Rb1, Rbl1/p107, and Rbl2/p130) in the auditory cells’ proliferation and emphasized our lack of information on their specific roles in the auditory system. We have previously demonstrated that lack of Rbl2/p130 moderately affects HCs’ and supporting cells’ (SCs) proliferation. Here, we present evidence supporting multiple roles for Rbl1/p107 in the developing and mature mouse organ of Corti (OC). Like other pRBs, Rbl1/p107 is expressed in the OC, particularly in the Hensen’s and Deiters’ cells. Moreover, Rbl1/p107 impacts maturation and postmitotic quiescence of HCs and SCs, as evidenced by enhanced numbers of these cells and the presence of dividing cells in the postnatal Rbl1/p107−/− OC. These findings were further supported by microarray and bioinformatics analyses, suggesting downregulation of several bHLH molecules, as well as activation of the Notch/Hes/Hey signaling pathway in homozygous Rbl1/p107 mutant mice. Physiological assessments and detection of ectopic HC marker expression in postnatal spiral ganglion neurons (SGNs) provided evidence for incomplete cell maturation and differentiation in Rbl1/p107−/− OC. Collectively, the present study highlights an important role for Rbl1/p107 in OC cell differentiation and maturation, which is distinct from other pRBs

    Solstice: An Electronic Journal of Geography and Mathematics: Vol. 31, No. 1

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    Peer Reviewedhttps://deepblue.lib.umich.edu/bitstream/2027.42/155623/1/SolsticeVolumeXXXINumber1.pdfhttps://deepblue.lib.umich.edu/bitstream/2027.42/155623/4/vitaFull2020.pdfDescription of SolsticeVolumeXXXINumber1.pdf : Solstice, Vol. XXXI, No. 1.Description of vitaFull2020.pdf : CVarlinghau
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