Highly Thermal Stable Broadband Near-Infrared Luminescence in Ni²⁺-Doped LaAlO₃ with Charge Compensator

Ni2+-doped LaAlO3 perovskite ceramic samples with a charge compensator (M4+) were prepared and their luminescent properties were investigated. The LaAlO3:Ni2+–M4+ (M = Sn, Hf, Ti, Zr) perovskites show a broad near-infrared (NIR) luminescence peaking at around 1070 nm with 150 nm fwhm due to the Ni2+:3T2-3A2 transition. The Sn4+-codoped one showed the highest NIR luminescence intensity. The valence state of major Ni ions is changed from Ni3+ to Ni2+ by codoping with Sn4+ based on X-ray absorption spectroscopy. The LaAlO3:Ni2+–Sn4+ shows the shortest luminescence peak wavelength (λem = 1070 nm) at 300 K due to the strong crystal field and also shows the highest quenching temperature, T50% of 608 K in the temperature dependence of PL intensity, due to the largest activation energy among the Ni2+-doped NIR phosphors ever reported. Based on the positive trend in the plot of T50% vs PL peak energy, it is concluded that the dominant quenching process in almost all Ni2+ phosphors is the thermally activated crossover process. It is also demonstrated that the luminescence peak wavelength is shifted from 1070 to 1235 nm continuously by substituting Ga ions for the Al site

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-6

Ved from August to December 2006. The darkest color shows August and the lightest color shows December.<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-2

Asm of a neuron (an arrow) and nerve axons (arrow heads). Immunohistochemistry. Bar = 0.1 mm<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-3

Rophages (an arrow). Immunohistochemistry. Bar = 0.1 mm<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-1

Ds point out the glial nodules. Hematoxylin and eosin stain. Bar = 0.5 mm<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-0

Ved from August to December 2006. The darkest color shows August and the lightest color shows December.<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan-5

Gned, and a phylogenetic tree was constructed by the neighbor-joining method. The corresponding nucleotide sequence of Aino virus strain JaNAr28 in the genus was used as an outgroup to root the tree. The percentage bootstrap values calculated from 1000 replications are indicated above the internal nodes. The scale represents the 10% nucleotide sequence divergence. The geographical origin and year of isolation of the strains are given in the parentheses.<p><b>Copyright information:</b></p><p>Taken from "Bovine epizootic encephalomyelitis caused by Akabane virus in southern Japan"</p><p>http://www.biomedcentral.com/1746-6148/4/20</p><p>BMC Veterinary Research 2008;4():20-20.</p><p>Published online 13 Jun 2008</p><p>PMCID:PMC2443122.</p><p></p

Bend Vibration of Surface Water Investigated by Heterodyne-Detected Sum Frequency Generation and Theoretical Study: Dominant Role of Quadrupole

Heterodyne-detected vibrational sum frequency generation spectroscopy was applied to the water surface for measuring the imaginary part of second-order nonlinear susceptibility (Im χ⁽²⁾) spectrum in the bend frequency region for the first time. The observed Im χ⁽²⁾ spectrum shows an overall positive band around 1650 cm^–1, contradicting former theoretical predictions. We further found that the Im χ⁽²⁾ spectrum of NaI aqueous solution exhibits an even larger positive band, which is apparently contrary to the flip-flop orientation of surface water. These unexpected observations are elucidated by calculating quadrupole contributions beyond the conventional dipole approximation. It is indicated that the Im χ⁽²⁾ spectrum in the bend region has a large quadrupole contribution from the bulk water

Clinical features of patients at the second liver biopsy after SVR.

<p>Clinical features of patients at the second liver biopsy after SVR.</p

Morphometric analysis.

<p>Morphometric analysis of Sirius red-positive (A) and α-SMA-positive (B) areas in SVR patients, with and without HCC. Positive areas for Sirius red and α-SMA immunohistochemistry were determined using the analysis application BZ-H3A (Keyence, Osaka, Japan). Note that both the Sirius red- and α-SMA-positive areas were not improved in the SVR-HCC group. CYGB and α-SMA-positive cells were counted under a × 100 objective. Note that the CYGB and α-SMA-positive cells numbers remained in the SVR-HCC group; * p < 0.05; **, p < 0.01.</p