Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancers

Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancer

Additional file 2 of Global DNA methylation profiling reveals chromosomal instability in IDH-mutant astrocytomas

Additional file 2. Fig. 2 Kaplan-Meier analysis of the full test cohort demonstrating significant differences in progression-free (A) and overall survival (B) between cases stratified by 2021 WHO grad

Additional file 3 of Global DNA methylation profiling reveals chromosomal instability in IDH-mutant astrocytomas

Additional file 3. Table 1 List of IDH-mutant astrocytoma case

Supplementary Figure from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancers

Supplementary Figure from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancer

Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancers

Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancer

Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancers

Supplementary Table from Integrated Metabolic Profiling and Transcriptional Analysis Reveals Therapeutic Modalities for Targeting Rapidly Proliferating Breast Cancer

Additional file 1 of Global DNA methylation profiling reveals chromosomal instability in IDH-mutant astrocytomas

Additional file 1. Fig. 1 Copy number variation differences between initial biopsies and tumor recurrences in IDH-mutant and IDH-wildtype astrocytomas (A) and IDH-mutant astrocytomas with known CIN and CS status (B

Supplementary Table S5 from Cells Lacking the <i>RB1</i> Tumor Suppressor Gene Are Hyperdependent on Aurora B Kinase for Survival

Supplementary Table S5 shows RNA sequencing data and gene set enrichment analysis of NCI-H82 cells Dox-On pRB cells grown in the presence or absence of DOX and then treated with DMSO or AZD2811.</p

Supplementary Table S2 from Cells Lacking the <i>RB1</i> Tumor Suppressor Gene Are Hyperdependent on Aurora B Kinase for Survival

Supplementary Table S2 shows Raw NGS reads and Log2 Norm Scores from Illumina sequencing of the pRB synthetic lethal screen.</p

Supplementary Table S6 from Cells Lacking the <i>RB1</i> Tumor Suppressor Gene Are Hyperdependent on Aurora B Kinase for Survival

Supplementary Table S6 shows mutational status of RB1 and TP53 and amplification status of MYC in cell line xenograft and PDX SCLC mouse models.</p