Supplementary Figure 3 from δ-Catenin Promotes Bevacizumab-Induced Glioma Invasion

Bevacizumab treatment led to increase in invasive and high expression of Î'-catenin. Silencing of Î'-catenin decreased bevacizumab-induced glioma invasion and Î'-catenin expression. Overexpressed Î'-catenin tumor cells showed more invasiveness and expressed high level ofÎ'-catenin protein in the tumor area in vivo.</p

Supplementary Figure 4 from δ-Catenin Promotes Bevacizumab-Induced Glioma Invasion

WST-1 assay and two-chamber assay</p

Supplementary Figure 1 from δ-Catenin Promotes Bevacizumab-Induced Glioma Invasion

Glioma cell cytotoxicity with bevacizumab treatment in vitro</p

Supplementary Figure 2 from δ-Catenin Promotes Bevacizumab-Induced Glioma Invasion

Bevacizumab increased the rate of wound closure</p

Supplementary Figure 3 from Oncolytic Herpes Virus Armed with Vasculostatin in Combination with Bevacizumab Abrogates Glioma Invasion via the CCN1 and AKT Signaling Pathways

Supplementary Figure 3 shows Full scans of the western blotting shown in the figure 6.</p

Additional file 10 of Annexin A2–STAT3–Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma

Additional file 10: Supplementary Figure S8. Effect of ANXA2 and OSMR knockdown on STAT3 and ERK signaling in U87ΔEGFR cells

Additional file 9 of Annexin A2–STAT3–Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma

Additional file 9: Supplementary Figure S7. Control of GBM cell proliferation and invasion by ANXA2 and OSMR

Additional file 5 of Annexin A2–STAT3–Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma

Additional file 5: Supplementary Figure S3. Correlations between mRNA expression of ANXA2 and 15 genes associated with the high angiogenesis–invasion phenotype (OSMR is not shown)

Additional file 7 of Annexin A2–STAT3–Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma

Additional file 7: Supplementary Figure S5. OSMR alone modulates the mesenchymal transition of GBM cells in vitro

Additional file 4 of Annexin A2–STAT3–Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma

Additional file 4: Supplementary Figure S2. GSEA enrichment plots of angiogenesis and invasion signatures in the four GBM subtypes