66 research outputs found
Party Groups and Policy Positions in the European Parliament
As the legislative body of the European Union, the European Parliament groups 732 elected representatives from over 170 national political parties from 25 member states. At the EP level, these members are affiliated with seven major party groups representing distinct policy positions. In this paper we provide precise estimates of these policy positions based on expert surveys in addition to characterizing the dimensionality of policy competition in the EP. Our results suggest not only that party groups have identifiable and differentiated positions on multiple issues of policy, but also that these positions group broadly into two orthogonal dimensions: one consisting of classic left-right social and economic issues, and the other related to the powers and scope of EU institutions.Party Competition, Policy Positions, European Parliament, Expert Surveys.
Committees and party cohesion in the European parliament
'Wie sichern politische Parteien in sich neu herausbildenden gesetzgebenden Körperschaften Parteidisziplin und -kohÀsion? In etablierten parlamentarischen Demokratien besitzen Parteien hÀufig ein so hohes Niveau an Einheitlichkeit, dass der Frage, wie und warum eine solche Parteidisziplin entsteht, bis zuletzt wenig Aufmerksamkeit geschenkt wurde. In sich neu herausbildenden gesetzgebenden Körperschaften ist die Transformation von rudimentÀren Parteiorganisationen in disziplinierte Parteien jedoch keine zwangslÀufige. Dieser Artikel untersucht, ob die politischen Gruppen im EuropÀischen Parlament (EP) Versuche unternehmen, Parteidisziplin durchzusetzen. Spezifischer untersucht der Beitrag, ob MEPs, die laufend gegen die eigene Partei stimmen, von dieser im Rahmen des Prozesses der Ausschussentsendung im EP bestraft werden.' (Autorenreferat)'How do political parties enforce party discipline and promote cohesiveness in newly emerging legislatures? Political parties in established parliamentary democracies typically exhibit such high levels of unity that the question of how and why such discipline arises has, until recently, received little attention. But in emerging legislatures the process of transforming rudimentary party organisations into disciplined parties is not inevitable. This article examines if the political groups in the European Parliament (EP) attempt to enforce party discipline. More specifically, the article asks the question if MEPs who consistently vote against the party are punished in terms of their committee assignments.' (author's abstract
Legislative Politics as Normal? Voting Behaviour and Beyond in the European Parliament
Committee Representation in the European Parliament
The European Parliament (EP) possesses a highly specialized committee system, operating in a complex institutional and political environment, yet little empirical work has investigated how MEPs are assigned to EP committees and what consequences this process has for representation and policy-making. In this article I examine the growth of EP committees and committee membership since 1979, and address the question of whether these committees are representative of the EP as a whole. Using an original data set of committee membership, national and EP party affiliation, MEP characteristics, and MEP policy preferences derived from roll-call votes, I address three key questions: Does committee membership reflect the party group composition of the EP? Do committee members possess specialized expertise in their committeesâ policy areas? And, finally, do committee membersâ general or committee-specific policy preferences differ substantially from those of the overall Parliament? The results suggest very strongly that, although committee members do tend to possess policy-specific expertise, committees are, nonetheless, highly representative of the EP as a whole, in terms of both party and policy representation
Dissecting the Shared Genetic Architecture of Suicide Attempt, Psychiatric Disorders, and Known Risk Factors
Background Suicide is a leading cause of death worldwide, and nonfatal suicide attempts, which occur far more frequently, are a major source of disability and social and economic burden. Both have substantial genetic etiology, which is partially shared and partially distinct from that of related psychiatric disorders. Methods We conducted a genome-wide association study (GWAS) of 29,782 suicide attempt (SA) cases and 519,961 controls in the International Suicide Genetics Consortium (ISGC). The GWAS of SA was conditioned on psychiatric disorders using GWAS summary statistics via multitrait-based conditional and joint analysis, to remove genetic effects on SA mediated by psychiatric disorders. We investigated the shared and divergent genetic architectures of SA, psychiatric disorders, and other known risk factors. Results Two loci reached genome-wide significance for SA: the major histocompatibility complex and an intergenic locus on chromosome 7, the latter of which remained associated with SA after conditioning on psychiatric disorders and replicated in an independent cohort from the Million Veteran Program. This locus has been implicated in risk-taking behavior, smoking, and insomnia. SA showed strong genetic correlation with psychiatric disorders, particularly major depression, and also with smoking, pain, risk-taking behavior, sleep disturbances, lower educational attainment, reproductive traits, lower socioeconomic status, and poorer general health. After conditioning on psychiatric disorders, the genetic correlations between SA and psychiatric disorders decreased, whereas those with nonpsychiatric traits remained largely unchanged. Conclusions Our results identify a risk locus that contributes more strongly to SA than other phenotypes and suggest a shared underlying biology between SA and known risk factors that is not mediated by psychiatric disorders.Peer reviewe
Schizophrenia and potentially preventable hospitalizations in the United States: a retrospective cross-sectional study
Culture and the Gender Gap in Competitive Inclination: Evidence from the Communist Experiment in China
The genetics of the mood disorder spectrum:genome-wide association analyses of over 185,000 cases and 439,000 controls
Background
Mood disorders (including major depressive disorder and bipolar disorder) affect 10-20% of the population. They range from brief, mild episodes to severe, incapacitating conditions that markedly impact lives. Despite their diagnostic distinction, multiple approaches have shown considerable sharing of risk factors across the mood disorders.
Methods
To clarify their shared molecular genetic basis, and to highlight disorder-specific associations, we meta-analysed data from the latest Psychiatric Genomics Consortium (PGC) genome-wide association studies of major depression (including data from 23andMe) and bipolar disorder, and an additional major depressive disorder cohort from UK Biobank (total: 185,285 cases, 439,741 controls; non-overlapping N = 609,424).
Results
Seventy-three loci reached genome-wide significance in the meta-analysis, including 15 that are novel for mood disorders. More genome-wide significant loci from the PGC analysis of major depression than bipolar disorder reached genome-wide significance. Genetic correlations revealed that type 2 bipolar disorder correlates strongly with recurrent and single episode major depressive disorder. Systems biology analyses highlight both similarities and differences between the mood disorders, particularly in the mouse brain cell-types implicated by the expression patterns of associated genes. The mood disorders also differ in their genetic correlation with educational attainment â positive in bipolar disorder but negative in major depressive disorder.
Conclusions
The mood disorders share several genetic associations, and can be combined effectively to increase variant discovery. However, we demonstrate several differences between these disorders. Analysing subtypes of major depressive disorder and bipolar disorder provides evidence for a genetic mood disorders spectrum
Bipolar multiplex families have an increased burden of common risk variants for psychiatric disorders.
Multiplex families with a high prevalence of a psychiatric disorder are often examined to identify rare genetic variants with large effect sizes. In the present study, we analysed whether the risk for bipolar disorder (BD) in BD multiplex families is influenced by common genetic variants. Furthermore, we investigated whether this risk is conferred mainly by BD-specific risk variants or by variants also associated with the susceptibility to schizophrenia or major depression. In total, 395 individuals from 33 Andalusian BD multiplex families (166 BD, 78 major depressive disorder, 151 unaffected) as well as 438 subjects from an independent, BD case/control cohort (161 unrelated BD, 277 unrelated controls) were analysed. Polygenic risk scores (PRS) for BD, schizophrenia (SCZ), and major depression were calculated and compared between the cohorts. Both the familial BD cases and unaffected family members had higher PRS for all three psychiatric disorders than the independent controls, with BD and SCZ being significant after correction for multiple testing, suggesting a high baseline risk for several psychiatric disorders in the families. Moreover, familial BD cases showed significantly higher BD PRS than unaffected family members and unrelated BD cases. A plausible hypothesis is that, in multiplex families with a general increase in risk for psychiatric disease, BD development is attributable to a high burden of common variants that confer a specific risk for BD. The present analyses demonstrated that common genetic risk variants for psychiatric disorders are likely to contribute to the high incidence of affective psychiatric disorders in the multiplex families. However, the PRS explained only part of the observed phenotypic variance, and rare variants might have also contributed to disease development
Gammelfleisch everywhere? public debate, variety of worldviews and regulatory change
Cultural theory has attracted considerable interest in the study of risk regulation. There has, however, been a lack of a systematic interest in its claims and in methodological issues. In this paper, we present seven claims that are either directly drawn from central claims of cultural theory or from complementary theories and assess them in the light of one single case, failure in meat inspections in Germany. These claims are assessed through the analysis of argumentation in newspapers
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