Fiber motion model based on yarn-forming mechanism in airflow

With the development of the textile industry, requirements relating to the quality of spinning of fibers to form yarn are becoming increasingly stringent. This paper analyzes the force of fibers in airflow to develop a drafting model and a twisted structural model of airflow, and numerically simulates the internal flow field of the drafting and twisting structures to obtain the optimal parameters for the model and nozzle. The structure is designed to improve the spinnability of different fibers. A fluid–solid coupling simulation is used to analyze the movement of fibers in the drafting channel, and parameters of the model are analyzed and optimized. The proposed model provides theoretical support for improving the speed and quality of spinning. </jats:p

Transport Phenomena Coupled by Chemical Reactions in Methane Reforming Ducts

Mass, heat and momentum transport processes are strongly coupled with catalytic chemical reactions in a methane steam reforming duct. In this paper, a three-dimensional calculation method is developed to simulate and analyze reforming reactions of methane, and the effects on various transport processes in a steam reforming duct. The results show that the design and operating parameters grouped as characteristic ratios have significant effects on the transport phenomen

Dioscin Ameliorates Polycystic Ovary Syndrome by Inhibiting PI3K/Akt Pathway-Mediated Proliferation and Apoptosis of Ovarian Granulosa Cells

To understand the mechanism underlying Dioscin inhibition of polycystic ovary syndrome, we have examined its effects on ovarian granulosa cells from letrozole-treated rats. To this end, Western blot was utilized to determine changes in the levels of Bcl-2, cleaved caspase-3, caspase-3, and phosphatidylinositol 3-kinase (PI3K)/protein kinase B pathway after Dioscin treatment in letrozole-treated rats. Dioscin ameliorated polycystic ovary syndrome by reducing the serum level of testosterone and increasing progesterone levels. It also inhibited proliferation and induced apoptosis of ovarian granulosa cells in the rat model by decreasing the level of Bcl-2 and elevating cleaved caspase-3. Western blot analysis revealed that Dioscin suppressed the PI3K/Akt pathway by inhibiting p-AKT/AKT. SC79, a p-AKT/AKT activator, reversed the effects of Dioscin on the proliferation and apoptosis of ovarian granulosa cells. In conclusion, Dioscin might present a novel therapeutic opportunity for patients with polycystic ovary syndrome.</p