A Perspective on Organic Agriculture in China - Opportunities and Challenges

With the rapid development of international production and trade in organic food, organic agriculture is also boosting in China. The milestone of Chinese organic agri-culture was set in 1990 with the first export of a certified organic product (tea) from Lin’an county of Zhejiang Province, China, which marked the launch of organic pro-duction in China. By the end of 2005, there had been about 4.384 million ha organic land, including 1.694 million ha organic, 0.61 million ha conversion as well as 2.08 million ha collection area, about half of which is certified area. About 4.93 million tons organic products and nearly 1600 projects had been certified with 300~400 varieties. According to the above data, now China ranks the 3rd largest country of organic production in the world. In China, organic development mostly is export oriented, the export products include beans, rice, tea, mushroom, vegetable, processed oil and herbs, etc. According to the certifying body COFCC of Ministry of Agriculture (MOA), the value of exported organic products increased from 0.3 million USD in 1995 to 0.35 billion USD at the end of the year 2004. Chinese domestic organic market started from 2000. Presently, most of the products sold in domestic markets are certified by COFCC and OFDC in some largest cities such as Beijing, Shanghai, Guangzhou, Nanjing and Shenzhen, etc. The price of the organic products is often up to 3 times the price of conventional products. Average organic food consumption accounts for 0.08% of the conventional food (LI 2006)

Histone demethylase JMJD1C is phosphorylated by mTOR to activate de novo lipogenesis.

Fatty acid and triglyceride synthesis increases greatly in response to feeding and insulin. This lipogenic induction involves coordinate transcriptional activation of various enzymes in lipogenic pathway, including fatty acid synthase and glycerol-3-phosphate acyltransferase. Here, we show that JMJD1C is a specific histone demethylase for lipogenic gene transcription in liver. In response to feeding/insulin, JMJD1C is phosphorylated at T505 by mTOR complex to allow direct interaction with USF-1 for recruitment to lipogenic promoter regions. Thus, by demethylating H3K9me2, JMJD1C alters chromatin accessibility to allow transcription. Consequently, JMJD1C promotes lipogenesis in vivo to increase hepatic and plasma triglyceride levels, showing its role in metabolic adaption for activation of the lipogenic program in response to feeding/insulin, and its contribution to development of hepatosteatosis resulting in insulin resistance

Irish consumers' perception of Chinese brands and how to improve the "Made in China" image

The Chinese government launched the “going abroad” policy in 2001 to encourage Chinese companies to invest and create Chinese brands in international markets. However, the perceptions of Chinese brands among Western consumers have been shown to be consistently low, especially amongst European consumers. This research aims to investigate the Irish consumers’ perception of Chinese brands and how to improve the “Made in China” image. This paper also investigates consumers’ perceptions and attitudes when making purchase decisions for Chinese brands based on gender, age, and nationality. The paper provides important recommendations for Chinese companies who plan to enter the European, and more particularly, the Irish market

Zc3h10 Acts as a Transcription Factor and Is Phosphorylated to Activate the Thermogenic Program.

Brown adipose tissue harbors UCP1 to dissipate chemical energy as heat. However, the transcriptional network that governs the thermogenic gene program is incompletely understood. Zc3h10, a CCCH-type zinc finger protein, has recently been reported to bind RNA. However, we report here that Zc3h10 functions as a transcription factor to activate UCP1 not through the enhancer region, but by binding to a far upstream region of the UCP1 promoter. Upon sympathetic stimulation, Zc3h10 is phosphorylated at S126 by p38 mitogen-activated protein kinase (MAPK) to increase binding to the distal region of the UCP1 promoter. Zc3h10, as well as mutant Zc3h10, which cannot bind RNA, enhances thermogenic capacity and energy expenditure, protecting mice from diet-induced obesity. Conversely, Zc3h10 ablation in UCP1+ cells in mice impairs thermogenic capacity and lowers oxygen consumption, leading to weight gain. Hence, Zc3h10 plays a critical role in the thermogenic gene program and may present future targets for obesity therapeutics