Supplemental Figures 1-5 and figure legends from Cell-Intrinsic Tumorigenic Functions of PPARγ in Bladder Urothelial Carcinoma

S1. Determination of PPARγ expression in UC and subtyping of UC cell lines. S2. Effect of pharmacological PPARγ inhibition on gene expression and cell cycle progression. S3. Validation of PPARG CRISPR KO and shRNA rescue experiment. S4. Validation of ChIP-seq data from the 5637 cell line. S5. PPARγ regulates proliferation and migration through SHH in UC.</p

Figure S2 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Figure S3 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Figure S4 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Supplementary Data_v1 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

Supplemental Method and Materials; Supplemental Figure Legends; Goes with the Longer version of the paper</p

Figure S6 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Figure S1 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Figure S5 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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Figure S7 from Metabolic Enzyme DLST Promotes Tumor Aggression and Reveals a Vulnerability to OXPHOS Inhibition in High-Risk Neuroblastoma

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