The Evolution of EU policy towards its CIS neighbours

When the EU began to plan enlargement, it was important to define and establish a new relationship with the successor states to the Soviet Union. This paper traces the evolution of the EU's policy towards those successor states from 1991 until the present. In the 1990s the EU tended to treat the post-Soviet states as if they were homogenous and policy was concentrated primarily on Russia. From 2000 onwards, a more differentiated policy was adopted, particularly once the European Neighbourhood Policy was launched. However, because of Russia's energy resources on which the EU was dependent, and because the EU's neighbourhood was also Russia's neighbourhood and Russian policy makers increasingly resented EU intervention into an area which they considered a vital sphere of Russia's interests, Russia still tended to dominate the attention of the EU.EU-CIS relations, EU-Russian relations, TACIS, European Neighbourhood Policy, EU and Central Asia

Nationalism and identity in the former Soviet Union

Nationalism is useful in consolidating state identity and bolstering state-building, but in the form of ethnic nationalism, it can undermine the integrity of the state. This paper examines the effect of non-Russian and Russian nationalism in the former Soviet Union. It finds that although Russian nationalism evokes more fear outside Russia than non-Russian nationalism, the violent conflicts in the former Soviet Union have been caused by the latter rather than the former. Non-Russian nationalism also threatens the integrity of the Russian Federation. The author concludes that although the Soviet Union was unique, the political use of nationalism anywhere following rapid change and economic dislocation can rapidly turn nationalism from a positive means of self-identification to a virulent ethnic exclusion of others which leads to conflict.Publisher's Versio

European Neighbourhood Policy and Economic Reforms in the Eastern Neighbourhood

The paper discusses the current and potential role of the European Neighbourhood Policy (ENP) in anchoring economic reforms in the countries of the EU's Eastern Neighbourhood. It claims that it is too early to assess the success of the ENP in this sphere especially given that the actual progress of the ENP agenda has been limited. A review of the empirical evidence on external reform anchors confirms that the ENP shares some features with the EU accession process that has proven to be an effective mechanism supporting major economic, political and social changes in the countries concerned. The eventual ENP economic offer is meaningful and integration with the EU is getting stronger public support in several CIS countries and among their political elites. On the other hand several factors limit the reform anchoring potential of the ENP. This paper offers recommendations on policies that could strengthen this potential

Mapping genomic loci implicates genes and synaptic biology in schizophrenia

Schizophrenia has a heritability of 60-80%1, much of which is attributable to common risk alleles. Here, in a two-stage genome-wide association study of up to 76,755 individuals with schizophrenia and 243,649 control individuals, we report common variant associations at 287 distinct genomic loci. Associations were concentrated in genes that are expressed in excitatory and inhibitory neurons of the central nervous system, but not in other tissues or cell types. Using fine-mapping and functional genomic data, we identify 120 genes (106 protein-coding) that are likely to underpin associations at some of these loci, including 16 genes with credible causal non-synonymous or untranslated region variation. We also implicate fundamental processes related to neuronal function, including synaptic organization, differentiation and transmission. Fine-mapped candidates were enriched for genes associated with rare disruptive coding variants in people with schizophrenia, including the glutamate receptor subunit GRIN2A and transcription factor SP4, and were also enriched for genes implicated by such variants in neurodevelopmental disorders. We identify biological processes relevant to schizophrenia pathophysiology; show convergence of common and rare variant associations in schizophrenia and neurodevelopmental disorders; and provide a resource of prioritized genes and variants to advance mechanistic studies

A telling story of IR in the periphery: Telling Turkey about the world, telling the world about Turkey

[No abstract available

Mapping genomic loci prioritises genes and implicates synaptic biology in schizophrenia

Schizophrenia has a heritability of 60–80%1, much of which is attributable to common risk alleles. Here, in a two-stage genome-wide association study of up to 76,755 individuals with schizophrenia and 243,649 control individuals, we report common variant associations at 287 distinct genomic loci. Associations were concentrated in genes that are expressed in excitatory and inhibitory neurons of the central nervous system, but not in other tissues or cell types. Using fine-mapping and functional genomic data, we identify 120 genes (106 protein-coding) that are likely to underpin associations at some of these loci, including 16 genes with credible causal non-synonymous or untranslated region variation. We also implicate fundamental processes related to neuronal function, including synaptic organization, differentiation and transmission. Fine-mapped candidates were enriched for genes associated with rare disruptive coding variants in people with schizophrenia, including the glutamate receptor subunit GRIN2A and transcription factor SP4, and were also enriched for genes implicated by such variants in neurodevelopmental disorders. We identify biological processes relevant to schizophrenia pathophysiology; show convergence of common and rare variant associations in schizophrenia and neurodevelopmental disorders; and provide a resource of prioritized genes and variants to advance mechanistic studies