MOESM1 of Transcriptomic and microstructural analyses in Liriodendron tulipifera Linn. reveal candidate genes involved in nectary development and nectar secretion

Additional file1: Table S1. List of 21 DEGs primers used for RT-qPCR

MOESM1 of Integrated intracellular metabolic profiling and pathway analysis approaches reveal complex metabolic regulation by Clostridium acetobutylicum

Additional file 1. All the metabolites detected in our study and the results of pathway analysis

Supplementary document for Optical wireless communication system performance in natural water turbulence of any strength - 6153492.pdf

Supplemental document

Grand average waveforms and topographic maps for L1 and L2 switch trials for the high and low inhibitory control (IC) group.

<p>Grand average waveforms and topographic maps for L1 and L2 switch trials for the high and low inhibitory control (IC) group.</p

Mean latencies (ms), language switch costs, and standard errors for the high and low inhibitory control (IC) group during the EEG experiment.

<p>Mean latencies (ms), language switch costs, and standard errors for the high and low inhibitory control (IC) group during the EEG experiment.</p

Grand average waveforms and topographic maps for L1 and L2 repeat trials for the high and low inhibitory control (IC) group.

<p>Grand average waveforms and topographic maps for L1 and L2 repeat trials for the high and low inhibitory control (IC) group.</p

Rk3 triggers G1 arrest in Eca109 and KYSE150 cells.

(A) Cell cycle distribution analyzed by flow cytometry. (B) The expression level of G1 transition-related proteins was evaluated by western blotting. *p p < 0.01 compared with control.</p

Anti-esophageal cancer effects of the ginsenoside Rk3 <i>in vitro</i> and <i>in vivo</i>.

(A) The chemical structure of the ginsenoside Rk3. (B) Cell viability was measured by the MTT assay. (C) Colony formation. (D) Representative image of the tumor. (E) Tumor volume. (F) Body weight of mice. (G) H&E staining of major organs and tumors. *p p p < 0.001 compared with control.</p

Rk3 induces autophagy in esophageal cancer cells.

(A) Autophagosomes were observed under TEM. Red arrows represent autophagosomes (scale bar = 1 μm). (B) Effect of different doses of Rk3 on the expression of autophagy-related proteins after treatment for 24 h. (C) Immunohistochemical staining of tumor tissue. (D) Cells were pretreated with 3-MA (5 mM) for 2 h and then incubated with the indicated dose of Rk3 for 24 h. The inhibition of cell proliferation was measured by MTT assays. (E) Cells were treated as state above, and Atg5, p62, LC3, Bax, c-Casp 3 and c-Casp 9 were analyzed by western blotting. *p p #p ##p < 0.01 compared with the Rk3-treated group.</p

Rk3 suppresses the PI3K/Akt/mTOR pathway in Eca109 and KYSE150 cells.

(A) Cells were treated with differernt concentrations of Rk3 for 24 h, and the expression of PI3K, p-PI3K, Akt, p-Akt, mTOR and p-mTOR was determined by western blotting. (B) Immunohistochemical staining of tumor tissue. *p p < 0.01 compared with the control.</p