18 research outputs found

    Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579-4

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    He wild-type with their proposed functions and between brackets the expression in the ccpA deletion strain compared to the wild-type in the early- and mid-exponential growth phase, respectively. Putative CRE-sites are indicated in front of the operon, the open arrow in the flow scheme stands for transport into the cell, whereas the closed arrows stand for reactions. L-Asp stands for L-Aspartate and P stands for phosphate.<p><b>Copyright information:</b></p><p>Taken from "Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579"</p><p>http://www.biomedcentral.com/1471-2180/8/62</p><p>BMC Microbiology 2008;8():62-62.</p><p>Published online 16 Apr 2008</p><p>PMCID:PMC2358912.</p><p></p

    Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579-5

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    encoding Nhe and Hbl of ATCC 14579. Ratios of gene regulation are given as found in the deletion strain compared to the wild-type, for the four different growth phases, early- and mid-exponential, transition and stationary phase, respectively. Closed arrows indicate the approximate position of the identified CRE-site, open arrows indicate binding sites for the pleiotropic regulator PlcR [59].<p><b>Copyright information:</b></p><p>Taken from "Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579"</p><p>http://www.biomedcentral.com/1471-2180/8/62</p><p>BMC Microbiology 2008;8():62-62.</p><p>Published online 16 Apr 2008</p><p>PMCID:PMC2358912.</p><p></p

    Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579-0

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    (black) and the deletion strain (grey) in BHI. Arrows in indicate sampling points at ODof 0.2 (early-exponential), 0.8 (mid-exponential), 4.0 (transition) and 8.0 (stationary), for both glucose determination and transcriptional analysis. Experiments shown are representative for all performed experiments.<p><b>Copyright information:</b></p><p>Taken from "Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579"</p><p>http://www.biomedcentral.com/1471-2180/8/62</p><p>BMC Microbiology 2008;8():62-62.</p><p>Published online 16 Apr 2008</p><p>PMCID:PMC2358912.</p><p></p

    Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579-6

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    (black) and the deletion strain (grey) in BHI. Arrows in indicate sampling points at ODof 0.2 (early-exponential), 0.8 (mid-exponential), 4.0 (transition) and 8.0 (stationary), for both glucose determination and transcriptional analysis. Experiments shown are representative for all performed experiments.<p><b>Copyright information:</b></p><p>Taken from "Assessment of CcpA-mediated catabolite control of gene expression in ATCC 14579"</p><p>http://www.biomedcentral.com/1471-2180/8/62</p><p>BMC Microbiology 2008;8():62-62.</p><p>Published online 16 Apr 2008</p><p>PMCID:PMC2358912.</p><p></p

    Positive role of cell wall anchored proteinase PrtP in adhesion of lactococci-1

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    <p><b>Copyright information:</b></p><p>Taken from "Positive role of cell wall anchored proteinase PrtP in adhesion of lactococci"</p><p>http://www.biomedcentral.com/1471-2180/7/36</p><p>BMC Microbiology 2007;7():36-36.</p><p>Published online 2 May 2007</p><p>PMCID:PMC1876236.</p><p></p

    Positive role of cell wall anchored proteinase PrtP in adhesion of lactococci-0

    No full text
    <p><b>Copyright information:</b></p><p>Taken from "Positive role of cell wall anchored proteinase PrtP in adhesion of lactococci"</p><p>http://www.biomedcentral.com/1471-2180/7/36</p><p>BMC Microbiology 2007;7():36-36.</p><p>Published online 2 May 2007</p><p>PMCID:PMC1876236.</p><p></p

    Hyper-spreading phenotype of a <i>srtA</i> mutant of <i>S. epidermidis</i> 1457.

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    <p>Spreading motility of <i>S. epidermidis</i> 1457 (WT), a <i>srtA</i> mutant derivative of this strain (<i>srtA</i>), and a complemented derivative of the <i>srtA</i> mutant (<i>srtA<sup>Se</sup></i>-pCN51) was assayed as described for <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0044646#pone-0044646-g001" target="_blank">Figure 1</a>.</p

    Hyper-spreading phenotype of <i>srtA</i> mutant <i>S. aureus</i> strains.

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    <p>From an overnight culture, an aliquot of 2 ┬Ál was spotted in the middle of a TSA plate, which was then incubated overnight at 37┬░C. The analyses include the laboratory strains <i>S. aureus</i> SH1000 and NCTC8325 (both labeled WT), as well as their <i>srtA</i> mutant derivatives (labeled <i>srtA</i>) and <i>srtA</i> mutants complemented with a plasmid pCN51-borne copy of <i>S. aureus srtA</i> (labeled <i>srtA</i>-pCN51). The spreading areas of the investigated mutant and parental strains were determined by ImageJ. The graphs show the areas covered in arbitrary units (AU) and respective standard deviations.</p

    The influence of <i>fnbpA</i>, <i>fnbpB</i>, <i>clfA</i> and <i>clfB</i> mutations on colony spreading of <i>S. aureus.</i>

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    <p>Spreading motility of <i>S. aureus</i> SH1000-derived <i>fnbpA</i>, <i>fnbpB</i>, <i>clfA</i> and/or <i>clfB</i> mutant strains or the <i>S. aureus</i> Newman <i>srtA</i> mutant strain was assayed as described for <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0044646#pone-0044646-g001" target="_blank">Figure 1</a>.</p
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