Additional file 2 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 2

Additional file 4 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 4

Additional file 5 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 5

Additional file 6 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 6

Additional file 1 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 1

Additional file 3 of Genome-wide analysis of the P450 gene family in tea plant (Camellia sinensis) reveals functional diversity in abiotic stress

Additional file 3

The model receiver operating characteristic (ROC) curve for the definitive NASH diagnosis in NAFLD patients.

<p>The model receiver operating characteristic (ROC) curve for the definitive NASH diagnosis in NAFLD patients.</p

CK-18 fragments are significantly higher in the serum with NASH compared to patients with non-NASH.

<p>The vertical axis represents serum CK-18 levels in U/L, whereas the horizontal axis refers to patient groups. The box and bars represent the interquartile range (the 25^th and 75^th percentiles) from the median (the horizontal line) and the 95% confidence interval, respectively. CK-18 fragments levels significantly increased in patients with NASH compared to non-NASH patients (median (Q25, Q75): 372.9 U/L (319.6, 431.4), 248.1 U/L (237.5, 266.6), respectively; <i>P</i> < 0.001).</p

RandomPhases.zip

This zip contains the three random phases with 1024*1024 pixels used in the simulations. This data is refrenced in our paper " Generation of non-iterative phase-only hologram based on hybrid phase mask"

Sudan IV staining of hepatic tissues in each group of rats.

<p>Hepatic steatosis emerged until 8^th week and aggravated gradually with the consumption of high-fat diet (B). Panacinar steatosis was observed at the end of 24^th week (C). These changes were improved in the liver of simvastatin-treated rats (D). A: Control ×200; B: 16^th week ×200; C: 24^th week ×200; D: Simvastatin-treated group ×200.</p