Additional file 1 of Non-canonical antigens are the largest fraction of peptides presented by MHC class I in mismatch repair deficient murine colorectal cancer

Additional file 1. Supplementary figures S1-S9

Supplementary Data 1 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS^G12C Inhibition in <i>KRAS^G12C</i>-mutant Colorectal Cancer

antibodies</p

Supplementary Figures S1-S6 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS^G12C Inhibition in <i>KRAS^G12C</i>-mutant Colorectal Cancer

Supplementary figures</p

Supplementary Data 2 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS^G12C Inhibition in <i>KRAS^G12C</i>-mutant Colorectal Cancer

concentrations of drugs</p

Supplementary Materials and Methods SM1 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS^G12C Inhibition in <i>KRAS^G12C</i>-mutant Colorectal Cancer

Supplementary materials and methods</p

Supplementary Table from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cancer

Supplementary Table from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cance

Supplementary Table from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cancer

Supplementary Table from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cance

Supplementary Figure from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cancer

Supplementary Figure from Targeting the DNA Damage Response Pathways and Replication Stress in Colorectal Cance

Supplementary Figure from Temozolomide Treatment Alters Mismatch Repair and Boosts Mutational Burden in Tumor and Blood of Colorectal Cancer Patients

Supplementary Figure from Temozolomide Treatment Alters Mismatch Repair and Boosts Mutational Burden in Tumor and Blood of Colorectal Cancer Patient