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PLZF induces an intravascular surveillance program mediated by long-lived LFA-1–ICAM-1 interactions

By Seddon Y. Thomas, Seth T. Scanlon, Klaus G. Griewank, Michael G. Constantinides, Adam K. Savage, Kenneth A. Barr, Fanyong Meng, Andrew D. Luster and Albert Bendelac

Abstract

PLZF-expressing NKT cells establish residence at intravascular locations, failing to enter the circulation because of constitutive interactions with LFA-1 and ICAM-1

Topics: Article
Publisher: The Rockefeller University Press
OAI identifier: oai:pubmedcentral.nih.gov:3173247
Provided by: PubMed Central

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