Article thumbnail

The hippocampal fimbria of cuprizone-treated animals as a structure for studying neuroprotection in multiple sclerosis

By M. Kipp, A. Norkus, B. Krauspe, T. Clarner, K. Berger, P. van der Valk, S. Amor and C. Beyer
Topics: Short Communication
Publisher: SP Birkhäuser Verlag Basel
OAI identifier:
Provided by: PubMed Central

To submit an update or takedown request for this paper, please submit an Update/Correction/Removal Request.

Suggested articles


  1. A histological histochemical and biochemical study of the macroscopically normal white matter in multiple sclerosis.
  2. A magnetization transfer imaging study of normal-appearing white matter in multiple sclerosis.
  3. (2010). Activated microglia mediate axoglial disruption that contributes to axonal injury in multiple sclerosis.
  4. Alternative activation of macrophages.
  5. (2009). Cuprizone effect on myelination, astrogliosis and microglia attraction in the mouse basal ganglia. Brain Res.
  6. Cuprizone treatment induces demyelination and astrocytosis in the mouse hippocampus.
  7. (1998). Imaging axonal damage of normal-appearing white matter in multiple sclerosis.
  8. (2010). In toxic demyelination oligodendroglial cell death occurs early and is FAS independent. Neurobiol Dis.
  9. (2008). Normal-appearing white matter in multiple sclerosis is in a subtle balance between inflammation and neuroprotection.
  10. (2010). Protective effects of microglia in multiple sclerosis.
  11. (2001). Quantitative contrast-enhanced magnetic resonance imagingtoevaluateblood–brainbarrierintegrityinmultiple sclerosis: a preliminary study. Mult Scler.
  12. Region-specific expression of immunoregulatory proteins on microglia in the healthy CNS.
  13. Regulation of innate immune responses in the brain.
  14. The cuprizone animal model: new insights into an old story.
  15. Vitamin E induces ramification and downregulation of adhesion molecules in cultured microglial cells.