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Atypical protein kinase C controls sea urchin ciliogenesis

By Gérard Prulière, Jacky Cosson, Sandra Chevalier, Christian Sardet and Janet Chenevert


The distribution and function of aPKC are examined during sea urchin ciliogenesis. The kinase concentrates in a ring at the transition zone between the basal body and the elongating axoneme. Inhibition of aPKC results in mislocalization of the kinase and defective ciliogenesis. Thus aPKC controls the growth of motile cilia in invertebrate embryos

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Publisher: The American Society for Cell Biology
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Provided by: PubMed Central
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