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Atypical protein kinase C controls sea urchin ciliogenesis

By Gérard Prulière, Jacky Cosson, Sandra Chevalier, Christian Sardet and Janet Chenevert

Abstract

The distribution and function of aPKC are examined during sea urchin ciliogenesis. The kinase concentrates in a ring at the transition zone between the basal body and the elongating axoneme. Inhibition of aPKC results in mislocalization of the kinase and defective ciliogenesis. Thus aPKC controls the growth of motile cilia in invertebrate embryos

Topics: Articles
Publisher: The American Society for Cell Biology
OAI identifier: oai:pubmedcentral.nih.gov:3113769
Provided by: PubMed Central
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