Hyperosmotic Stress-Induced Corneal Epithelial Cell Death through Activation of Polo-like Kinase 3 and c-Jun
High extracellular osmotic pressure causes stress to corneal epithelial wound healing. The present study demonstrates a novel signaling mechanism in addition to stress-induced MAPK pathways in human corneal epithelial cells to regulate the AP-1 transcription complex through the activation of Plk3, resulting in changes of cell fate
Publisher: Association for Research in Vision and Ophthalmology, Inc.
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