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Molecular allelokaryotyping of T-cell prolymphocytic leukemia cells with high density single nucleotide polymorphism arrays identifies novel common genomic lesions and acquired uniparental disomy

By Daniel Nowak, Emilie Le Toriellec, Marc-Henri Stern, Norihiko Kawamata, Tadayuki Akagi, Martin J. Dyer, Wolf-Karsten Hofmann, Seishi Ogawa and H. Phillip Koeffler

Abstract

T-cell prolymphocytic leukemia is a rare aggressive lymphoproliferative disease with a mature T-cell phenotype and characteristic genomic rearrangements of 14q11. This study identifies numerous new potential target genes in common breakpoints, deletions and regions of acquired uniparental disomy

Topics: Original Article
Publisher: Ferrata Storti Foundation
OAI identifier: oai:pubmedcentral.nih.gov:2663615
Provided by: PubMed Central
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