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The possible roles of histamine, 5-hydroxytryptamine and prostaglandin F2α as mediators of the acute pulmonary effects of endotoxin

By J.R. Parratt and R.M. Sturgess

Abstract

1 In an attempt to investigate the possible role of released vasoactive substances in mediating the pulmonary pressor responses to E. coli endotoxin, cats were pretreated with histamine, 5-hydroxytryptamine (5-HT) or prostaglandin antagonists, with a histamine depleting agent (compound 48/80) or with an inhibitor of prostaglandin synthetase (sodium meclofenamate)

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OAI identifier: oai:pubmedcentral.nih.gov:1667448
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