Mice immunized against B.rodhaini by means of a drug-controlled
infection were subsequently resistant to infection with B.microti
and B.ratti. In the reciprocal experiments the protection against
B.rodhaini was less effective. B.rodhaini immunized mice were also
considerably protected against P.vinckei infection, whereas protection
against P.berqhei did not occur. Antibody determinations
indicated that the heterologous protection cannot be explained by
the occurrence of cross-reacting antibodies. Because of similarity
with the non-specific suppression of babesiosis in BCG-infected
mice, the same effector mechanism is postulated to explain the
infection-induced homologous and heterologous protection. Unlike
non-specifically induced protection, the induction of acquired
resistance by means of a drug-controlled B.rodhaini infection is
thymus-dependent
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