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Tumor necrosis factor alpha acts as an autocrine second signal with gamma interferon to induce nitric oxide in group B streptococcus-treated macrophages.

By K J Goodrum, J Dierksheide and B J Yoder

Abstract

Nitric oxide production by mouse macrophages treated with group B streptococci and gamma interferon was inhibited by cytochalasin B or by antibody neutralization of macrophage-derived tumor necrosis factor alpha. Phagocytosis-induced tumor necrosis factor alpha is responsible for group B streptococcus-induced nitric oxide production in interferon-treated macrophages

Topics: Research Article
Year: 1995
OAI identifier: oai:pubmedcentral.nih.gov:173516
Provided by: PubMed Central
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