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Resistance to Autolysis in Vancomycin-Selected Staphylococcus aureus Isolates Precedes Vancomycin-Intermediate Resistance

By Susan Boyle-Vavra, Mamatha Challapalli and Robert S. Daum

Abstract

Four clinical U.S. glycopeptide intermediate resistant Staphylococcus aureus (GISA) isolates were resistant to Triton X-100-induced autolysis. Similar resistance was demonstrated in an isolate obtained after a single passage of a susceptible clinical isolate in low-level vancomycin. Strains with the vancomycin-induced Triton X-100 resistance phenotype produced active murein hydrolases but were resistant to lysis by murein hydrolases

Topics: Mechanisms of Resistance
Publisher: American Society for Microbiology
Year: 2003
DOI identifier: 10.1128/AAC.47.6.2036-2039.2003
OAI identifier: oai:pubmedcentral.nih.gov:155830
Provided by: PubMed Central
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