Abstract. Fluid-structure interaction describing wave propagation in arteries driven by the pulsatile blood flow is a complex problem. Whenever possible, simplified models are called for. One-dimensional models are typically used in arterial sections that can be approximated by the cylindrical geometry allowing axially symmetric flows. Although a good first approximation to the underlying problem, the one-dimensional model suffers from several drawbacks: the model is not closed (an ad hoc velocity profile needs to be prescribed to obtain a closed system) and the model equations are quasi-linear hyperbolic (oversimplifying the viscous fluid dissipation), typically producing shock wave solutions not observed in healthy humans. In this manuscript we derived a simple, closed reduced model that accounts for the viscous fluid dissipation to the leading order. The resulting fluid-structure interaction system is of hyperbolic-parabolic type. Arterial walls were modeled by a novel, linearly viscoelastic cylindrical Koiter shell model and the flow of blood by the incompressible, viscous Navier–Stokes equations. Kelvin–Voigt-type viscoelasticity was used to capture the hysteresis behavior observed in the measurements of the arterial stress-strain response. Using the a priori estimates obtained from an energy inequality, together with the asymptotic analysis and ideas from homogenization theory for porous media flows, we derived an effective model whic
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