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Response of alveolar macrophages to inhaled particulates

By Martina Dörger and Fritz Krombach
Topics: Medizin, ddc:610
Publisher: Ludwig-Maximilians-Universität München
Year: 2002
DOI identifier: 10.1159/000048887
OAI identifier: oai:epub.ub.uni-muenchen.de:16774
Provided by: Open Access LMU

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  1. (1999). Additional NO2 exposure induces a decrease in cytokine specific mRNA expression and cytokine release of particle and fibre exposed human alveolar macrophages.
  2. (2000). Allmeling A-M, Krombach F: Comparison of the phagocytic response of rat and hamster alveolar macrophages to man-made vitreous fibers in vitro. Hum Exp Toxicol
  3. (1984). Alterations in macrophage functions by environmental chemicals. Environ Health Perspect
  4. (1996). Antioxidant and redox regulation of gene transcription.
  5. (1991). AR: Differential proliferation of rat lung fibroblasts induced by the platelet-derived growth factorAA, -AB, and -BB isoforms secreted by rat alveolar macrophages.
  6. (1988). AR: Pulmonary macrophages are attracted to inhaled particles through complement activation. Exp Lung Res
  7. (1997). Castranova V: Comparison of inducible nitric oxide synthase gene expression and lung inflammation following intratracheal instillation of silica, coal, carbonyl iron, or titanium dioxide in rats.
  8. Cell size of alveolar macrophages: an interspecies comparison. Environ Health Perspect 1997;105(suppl 5):
  9. (1986). Cohn ZA: The alveolar macrophage.
  10. (1994). Current perspectives on particulate induced pulmonary tumours. Hum Exp Toxicol
  11. (1997). Cytokines and particle-induced inflammatory cell recruitment. Environ Health Perspect
  12. Dey RD: Enhancement of nitric oxide production by pulmonary cells following silica exposure. Environ Health Perspect 1998;106(suppl 5):
  13. (1990). DH: Pulmonary reaction to long and short asbestos fibers is independent of fibroblast growth factor production by alveolar macrophages.
  14. (1994). E: Asbestos fibers and interferon-gamma up-regulate nitric oxide production in rat alveolar macrophages.
  15. (1998). E: Asbestos inhalation induces reactive nitrogen species and nitrotyrosine formation in the lungs and pleura of the rat.
  16. Erdogdu G: Secretion of fibronectin by mineral dust-derived alveolar macrophages and activated peritoneal macrophages.
  17. (1997). Evidence that exposure of particulate air pollutants to human and rat alveolar macrophages leads to differential oxidative response.
  18. Free radical activity of industrial fibers: Role of iron in oxidative stress and activation of transcription factors.
  19. (1997). Hartsky MA: Inhalation of high concentrations of low toxicity dusts in rats results in impaired pulmonary clearance mechanisms and persistent inflammation. Toxicol Appl Pharmacol
  20. (1993). Hartsky MA: Role of alveolar macrophage chemotaxis and phagocytosis in pulmonary clearance responses to inhaled particles: Comparisons among rodent species. Microsc Res Tech
  21. (1991). Holian A: In vitro bioactivity of asbestos for the human alveolar macrophage and its modification by IgG.
  22. (2001). Hyperoxia upregulates the NO pathway in alveolar macrophages in vitro: Role of AP-1 and NF-kappaB.
  23. (1994). Influences of gender, species, and strain differences in pulmonary toxicological assessments of inhaled particles and/or fibers;
  24. (1992). Intracellular particle dissolution in alveolar macrophages. Environ Health Perspect
  25. (1998). K: In vitro study of human alveolar macrophages inflammatory mediator transcriptions and releases induced by soot FR 101, Printex 90, titandioxide and Chrysotile B.
  26. KB: The role of reactive oxygen and nitrogen species in the response of airway epithelium to particles. Environ Health Perspect
  27. Kobzik L: Alveolar macrophage interaction with air pollution particles. Environ Health Perspect 1997;105(suppl 5):1191–1195.
  28. (1999). Kobzik L: Role of the scavenger receptor MARCO in alveolar macrophage binding of unopsonized environmental particles.
  29. (1999). Kohyama N: Biological effects of manmade mineral fibers (I) – Reactive oxygen species production and calcium homeostasis in alveolar macrophages. Ind Health
  30. Krombach F: Differential responses of rat alveolar and peritoneal macrophages to man-made vitreous fibers in vitro.
  31. Krombach F: Expression of inducible nitric oxide synthase and formation of nitric oxide by alveolar macrophages: An interspecies comparison. Environ Health Perspect 1997;105(suppl 5):1297–
  32. Krombach F: Interspecies comparison of rat and hamster alveolar macrophage antioxidative and oxidative capacity. Environ Health Perspect 1997;105(suppl 5):
  33. (2001). Krombach F: Phenotypic and functional differences between rat alveolar, pleural, and peritoneal macrophages. Exp Lung Res
  34. (1996). MacNee W: Free radical activity and pro-inflammatory effects of particulate air pollution (PM10) in vivo and in vitro. Thorax
  35. (1990). Macrophages and polymorphonuclear neutrophils in lung defense and injury. Am Rev Respir Dis
  36. (1992). MC: Modulation of eicosanoid production by human alveolar macrophages exposed to silica in vitro. Environ Health Perspect
  37. (1987). Mossman BT: Generation of superoxide from alveolar macrophages exposed to asbestiform and nonfibrous particles. Cancer Res
  38. (1998). Mossman BT: Mechanisms of asbestos-induced nitric oxide production by rat alveolar macrophages in inhalation and in vitro models. Free Radic Biol Med
  39. Muhle H: Non-animal tests for evaluating the toxicity of solid xenobiotics.
  40. (1996). Nitric oxide (NO) synthase activity in the lung and NO synthesis in alveolar macrophages of rats increased on exposure to asbestos.
  41. (1996). Ouellet S: Distinctive profile of alveolar macrophage-derived cytokine release induced by fibrogenic and nonfibrogenic mineral dusts.
  42. (1997). Oxidative stress: Oxidants and antioxidants. Exp Physiol
  43. (1993). Particle opsonization and lung macrophage cytokine response. In vitro and in vivo analysis.
  44. PH: Superoxide anion release by alveolar macrophages exposed to respirable industrial fibers: Modifying effect of fibre opsonization. Exp Toxicol Pathol
  45. (1989). PM: Asbestos fibers and silica particles stimulate rat alveolar macrophages to release tumor necrosis factor. Autoregulatory role of leukotriene B4. Am Rev Respir Dis
  46. (1990). PM: Inhibition of alveolar macrophage cytotoxicity by asbestos: Possible role of prostaglandins.
  47. Pulmonary and thoracic macrophage subpopulations and clearance of particles from the lung. Environ Health Perspect
  48. (1989). Pulmonary inflammation and fibrosis in a murine model of asbestosis and silicosis. Possible role of tumor necrosis factor. Inflammation
  49. (1996). RB: Stimulation of human and rat alveolar macrophages by urban air particles: Effects on oxidant radical generation and cytokine production. Toxicol Appl Pharmacol
  50. (1998). Retention and intracellular distribution of instilled iron oxide particles in human alveolar macrophages.
  51. (1992). Role of the alveolar macrophage in lung injury: Studies with ultrafine particles. Environ Health Perspect
  52. (2000). Scheuch G: Clearance of particles deposited in the lungs; in Heyder J, Gehr P (eds): Particle Lung Interactions.
  53. Schlüter T: Mechanisms of particle-induced activation of alveolar macrophages. Toxicol Lett 1996;88:
  54. SG: Species variation in lung antioxidant enzyme activities.
  55. (1998). Smiling alveolar macrophage.
  56. (1997). Species differences in NO formation by rat and hamster alveolar macrophages in vitro.
  57. (1986). Stöber W: Chronic effects on the respiratory tract of hamsters, mice and rats after long-term inhalation of high concentrations of filtered and unfiltered diesel engine emissions.
  58. (1997). Use of mechanistic data in assessing human risks from exposure to particles. Environ Health Perspect
  59. (1996). Vogelmeier C: Characterization and quantification of alveolar monocytes in patients with chronic inflammatory lung disease.

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