Antifungal activity against the pathogen, Botrytis cinerea, and a bioassay
organism, Cladosporium cladosporioides, declined with advancing strawberry fruit
maturity as shown by thin layer chromatography (TLC) bioassays. Preformed
antifungal activity was also present in flower tissue. The fall in fruit
antifungal compounds was correlated with a decline in natural disease resistance
(NDR) against B. cinerea in-planta. Crude extracts of green stage I fruit (7
days after anthesis) contained at least two preformed antifungal compounds (Rf =
0.44 and 0.37) that were not present in white and red stage fruit. These
compounds were shown with TLC reagent sprays to be neither phenolics nor
alkaloids. Positive reactions to Ehrlich’s reagent suggested that Rf = 0.37 was
a terpene. Most antifungal activity was found in the achenes of green stage I
fruit. However, antifungal activity was found in all tissue types (viz. pith,
cortex, epidermis) of green stage I fruit. TLC bioassays revealed that all fruit
stages yielded antifungal activity at the origin (Rf = 0.00). The approximate
area of fungal inhibition at the origin in green stage 1 fruit extracts was
1.87-fold and 1.73-fold greater than in white and red stages, respectively. TLC
reagent sprays showed that the antifungal compound(s) at origin included
phenolics. This observation is consistent with previous reports that phenolic
compounds in strawberry fruit are inhibitory to B. ciner
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