LETTER ABO blood type B and


secretor status as genetic risk factors for chronic pancreatitis Sir, We thank Kirsten et al1 for their comments on our recent study,2 in which we reported a genome-wide analysis on ∼4000 volun-teers from the population-based Study of Health in Pommerania (SHIP) cohort3 with replication in 1444 blood donors. We had found a significant association between higher serum lipase activities and three single nucleotide polymorphisms (SNP) as well as the fucosyltransferase-2 (FUT2) non-secretor status (homozygosity for the FUT2 p.W134X mutation) and th

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